(08-18-2023, 07:57 PM)Sleepy Quixote Wrote: @Cam12

Hey Cam, PLM activity can be inferred in OSCAR charts, takes a bit of a trained eye to pick it up and until you really get it down trying to identify it amongst all the other noise can be a bit misleading. The best way to monitor PLM activity is with a sleep monitor that has an accelerometer built in. Preferably a monitor you can attach to various appendages, especially those known to be most active. In my case its most frequently the left foot. The one I use is the Sleepon Go2Sleep SE, it covers SPo2, heart rate, sleep quality and motion. The SE part is important because it includes finger tip holders that also work well on toes, the ring alone isn't stable enough on an appendage doing the hoki poki while we sleep. Unfortunately none of this particular devices data can be imported into OSCAR, I recommend it because its the best for tracking PLMs and its easy enough to sync up by putting it on at the same time you start the pap, then use the laptop to check OSCAR while correlating PLM events on a tablet or phone. (The Sleepon Windows app is a waste of time for PLM)

I'm attaching a screenshot of it's mobile output so you can check it out. The green dots populating the lower portion of the screen represent movement. (Be sure to scroll down to the bottom of the image to see the activity)

Thank you so much! So glad I asked this question. Going to get that device though my movements are primarily upper body, used to be mostly legs, feet but now hips, abdomen, shoulders, arms so if I can get it to attach and record movements well somewhere, done deal.I love that it shows intensity and sleep stages. I can see on the screenshot that each high intensity movement is followed by an increase in heart rate. Should be invaluable once I can sync that data, OSCAR (like respiratory rate) and sleep video. 

Don’t mean to worry you unnecessarily but a big reason I’m asking is that I had a recent brain MRI that shows deep periventricular hyperintensities (areas where white matter has been damaged by O2 deprivation and/or capillary “leaks”) and found one study (attached) that links PLMS to these, taking into account sleep apnea (AHI idex). Theoretically, they say that each PLMS spikes heart rate and esp. blood pressure by 20-30 (can’t recall measurement but however they measure blood pressure) and that these, over time, may lead to the weakening of capillary walls and these hyperintensities. As my first brain MRI and since many people get these hyperintensities as they age, it’s difficult to know for sure if that’s why I have them (age 58) but at least now have a baseline. My neurologist is minimizing it but if I can show I’m having these spikes (at least in heart rate, checked blood pressure monitors but best device is only 5/hr) when I have movements, then maybe it’ll change treatment course I.e.) she encourages me to not be too concerned as out how many movements I’m having (currently about 60/hr and severe EDS every day) but rather on “how you feel” but, if there’s an association I can prove, maybe she’ll focus on reducing movements.

A couple of questions if you don’t mind:
 
Do you find that the sleep stage data is accurate I.e) that more REM/N3 from monitor = less EDS?

How closely time-wise, can you sync device start with CPAP start? Any advice on how to do it so they’re as closely synced as possible? 

Is the Screenshot attachment from the Windows App or another app? I have a Windows laptop but all else Apple - iPads, iPhone . Hopefully there’s an iOS app but if not, would you still recommend the device, even though the windows app is sketchy?

Thanks again!

(08-18-2023, 08:01 PM)jwest Wrote: Pretty sure you should be able to line the O2 up with your PAP data. There is an explanation guide somewhere on this website.

(08-18-2023, 08:07 PM)Sleepy Quixote Wrote: @Cam12

jwest attached a pretty good example of what PLM looks like in an OSCAR chart, check it out. Mine are rarely that well defined but that's a good primer for you.